Looking for an Off Switch for Celiac Disease

Looking for an Off Switch for Celiac Disease:

Latest News on Celiac's Disease!

 New research published in the Feb. 23 issue of the Journal of Biological Chemistry identifies an enzyme that turns off transglutaminase 2, potentially paving the way for new treatments for celiac disease.

Newswise — Celiac disease is an autoimmune disorder that affects by some estimates nearly 1 in 100 people. Celiac disease symptoms are triggered by gluten, a protein found in wheat and related plants, but gluten doesn’t act alone to cause the digestive symptoms that patients suffer. Rather, gluten induces an overactive immune response when it’s modified by the enzyme transglutaminase 2, or TG2, in the small intestine. New research published in the Feb. 23 issue of the Journal of Biological Chemistry identifies an enzyme that turns off TG2, potentially paving the way for new treatments for celiac disease.

As Cells Age, the Fat Content Within Them Shifts

As Cells Age, the Fat Content Within Them Shifts

This is an interesting article about the change that takes place in the cells as they age.

Botox’s Sweet Tooth Underlies Its Key Neuron-Targeting Mechanism

Botox’s Sweet Tooth Underlies Its Key Neuron-Targeting Mechanism

This article struck me as one of interest  to those who are thinking of having Botox but also who have had this:


A study co-led by Rongsheng Jin, professor of physiology & biophysics at the University of California, Irvine; Min Dong with Boston Children’s Hospital-Harvard Medical School; and Andreas Rummel with the Hannover Medical School in Germany, reveals an important general mechanism by which the pathogen is attracted to, adapts to and takes advantage of glycan modifications in surface receptors to invade motor neurons. Glycans are chains of sugars synthesized by cells for their development, growth, functioning or survival. Results appear June 13 in Nature Structural and Molecular Biology.

  Read the full article by clicking the link above.